Atrioventricular Block (Nursing)

Anthony Kashou; Amandeep Goyal; Tran Nguyen; Intisar Ahmed; Lovely Chhabra

Atrioventricular Block (Nursing)

Learning Outcome

List the causes of heart block
Describe the presentation of heart block
Recall the types of heart block
Summarize the treatment of heart block

Introduction

Atrioventricular (AV) conduction is evaluated by assessing the relationship between the P waves and QRS complexes. Normally, there is a P wave that precedes each QRS complex by a fixed PR interval of 120 to 200 milliseconds. AV block represents a delayed electrical impulse from the atria to the ventricles. This can be due to an anatomical or functional impairment in the heart’s conduction system. This disruption in normal electrical activity can be transient or permanent. In general, there are three degrees of AV nodal blocks: first degree, second degree (Mobitz type 1 or 2), and third-degree.[1][2][3]

At this time, there is no well-characterized large study about the relationship between different types of AV block with age, race, or gender. AV block is sometimes seen in athletes and in patients with congenital heart disorders.

Nursing Diagnosis

Ineffective tissue perfusion
Risk for falls
Impaired cardiac function
Anxiety

Causes

Higher degrees of AV block often suggest some underlying pathology. This is known as a pathophysiologic AV block. About half of such cases are a result of chronic idiopathic fibrosis and sclerosis of the conduction system.

Another common source is ischemic heart disease which is responsible for around 40 percent of cases of AV block [4].

AV block is also associated with cardiomyopathies, including hypertrophic obstructive cardiomyopathy and infiltrative conditions such as sarcoidosis and amyloidosis. Infectious causes such as Lyme disease, rheumatic fever, endocarditis, viruses as well as autoimmune disease such as systemic lupus erythematosus should also be explored [5][6][7][8].

Other potential triggers include cardiac surgery, medications, and inherited conditions [9].

Risk Factors

First-degree AV block can originate from various locations within the conduction system. The levels of conduction delay include the atrium, AV node (most common in first-degree heart block), Bundle of His, bundle branches, fascicles, Purkinje system. Mobitz type I second-degree AV block usually occurs within the AV node, while Mobitz type II second-degree AV block mainly originates from conduction system disease below the level of the AV node (in the bundle of His and in the bundle branches). In third-degree AV block, no atrial impulses reach the ventricle- it can occur in the AV node or in the infranodal specialized conduction system. [10]

The following medications can affect different levels of conduction delay:

Increased parasympathetic tone, digoxin (which upgrades vagotonic action), calcium channel blockers (which obstruct the inward calcium current responsible for depolarization), and beta-blockers can affect the AV node
Medications such as procainamide, quinidine, and disopyramide can block sodium channels and delay conduction in the bundle of His
Similarly, though rarely, medications such as procainamide, quinidine, and disopyramide can also delay infra-Hisian conduction system

Assessment

History taking for patients with concerns for AV block should include:

History of heart disease, both congenital and acquired
Full list of medications and dosing. Particular drugs of interest include beta-blockers, calcium channel blockers, antiarrhythmic drugs, digoxin
Recent cardiac procedure
Signs and symptoms associated with other systemic diseases associated with heart block (amyloidosis, sarcoidosis)
Baseline exercise capacity
Potential exposure to tick bites

The following symptoms should raise concerns:

Dyspnea
Fatigue
Chest pain
Presyncope or syncope
Sudden cardiac arrest

Evaluation

First degree. In first-degree AV block, the P waves always precede the QRS complexes, but there is a prolongation of the PR interval. The PR interval will be greater than 200 milliseconds in duration without any dropped beats. There is a delay, without interruption, in conduction from the atrium to the ventricle. All atrial activation is eventually transmitted to the ventricles. The delay is typically due to a minor AV conduction defect occurring at or below the AV node.

Causes. There are multiple causes of first-degree AV block, including simply being a normal variant. Other causes include inferior myocardial infarction (MI), increased vagal tone (e.g., athletes), status post-cardiac surgery, myocarditis, hyperkalemia, or even medication-induced (e.g., beta-blockers, non-dihydropyridine calcium channel blocks, adenosine, digitalis, and amiodarone).
Clinical significance. This is benign and does not result in any hemodynamic instability. No specific treatment is required.

Second degree, Mobitz type 1 (Wenckebach). In second-degree Mobitz type 1 AV block, there is a progressive prolongation of the PR interval, which eventually culminates in a non-conducting P wave. The PR interval continues to prolong with each beat of the cycle, and the subsequent PR lengthening is progressively shorter. The PR interval before the dropped beat is the longest of the cycle, and the PR interval after the dropped beat is the shortest as the cycle starts over.

Mechanism. This is usually a result of a reversible conduction block at the level of the AV node. There is typically a functional suppression of AV conduction. The AV nodal cells seem to progressively fatigue until they fail to conduct an impulse to the ventricles and a dropped beat occurs.
Causes. There are multiple causes of second-degree Mobitz type 1 (Wenckebach) AV block, including reversible ischemia, myocarditis, increased vagal tone, status post-cardiac surgery, or even medications that slow AV nodal conduction (e.g., beta-blockers, non-dihydropyridine calcium channel blocks, adenosine, digitalis, and amiodarone).
Clinical significance. Differentiating between second-degree Mobitz type 1 (Wenckebach) and Mobitz type 2 AV blocks is important as the management and treatment are different. Mobitz type 1 is often a benign rhythm. Most patients are asymptomatic, and there tends to be a minimal hemodynamic disturbance. Patients that are asymptomatic do not require treatment and can be monitored on an outpatient basis. Patients that are symptomatic typically respond to atropine and rarely require permanent cardiac pacing. Medication-induced impairment of AV conduction is often reversible after stopping the offending agent.

Second degree, Mobitz type 2. In second-degree Mobitz type 2 AV block, there are intermittent non-conducted P waves without warning. Unlike Mobitz type 1 (Wenckebach), there is no progressive prolongation of the PR interval; instead, the PR interval remains constant, and the P waves occur at a constant rate with unchanged P-P intervals. Because the P waves continue to occur at normal intervals, the R-R interval surrounding the dropped beat is simply a multiple of the preceding R-R interval and remains unchanged.

Mechanism. In Mobitz type 2, the block occurs farther along the electrical conduction system below the AV node.
In this case, the cells abruptly and unpredictably fail to conduct an impulse from the atria to the ventricles. This is often the result of structural damage to the conduction system.
Because the defect occurs below the AV node and often times distal to the His Bundle, it produces wide, bizarre-appearing QRS complexes. In the remaining cases, the defect is located within the Bundle of His, resulting in the normal, narrow QRS complexes. There can be a fixed P:QRS relationship (e.g., 2:1, 3:1) or no pattern at all.
Causes. Common causes of second-degree Mobitz type 2 AV block include anterior MI, causing septal infarction of the bundle branches. Other causes include idiopathic fibrosis of the conducting system, autoimmune (e.g., systemic sclerosis or systemic lupus erythematosus) or inflammatory (e.g., myocarditis, Lyme disease, or rheumatic fever) conditions, infiltrative myocardial disease (hemochromatosis, sarcoidosis, or amyloidosis), electrolyte imbalance (e.g., hyperkalemia), medication-induced (e.g., beta-blockers, non-dihydropyridine calcium channel blockers, digitalis, adenosine, or amiodarone), or status post-cardiac surgery (e.g., mitral valve repair).
Clinical significance. Mobitz type 2 AV block can be associated with severe bradycardia and hemodynamic instability. It has a greater risk of progressing to third-degree (complete) heart block or asystole. Because the onset of dropped beats can occur abruptly and unexpectedly, hemodynamic instability and the consequential syncope and potentially sudden cardiac death can occur at any moment. Thus, patients require a permanent pacemaker. While Mobitz type 1 can improve with atropine, giving atropine in the setting of Mobitz type 2 can worsen the block and increase the risk of complete heart block or asystole.

Third-degree (complete). In third-degree, or complete, heart block there is an absence of AV nodal conduction, and the P waves are never related to the QRS complexes. If ventricular conduction occurs, it is maintained by a junctional or ventricular escape rhythm. There is a complete dissociation between the atria and ventricles and they conduct independently of each other. The P waves (atrial activity) are said to “march through” the QRS complexes at their regular, faster rate. The QRS complexes (ventricular activity) also occur at a regular, but slower rate. There are two independent rhythms occurring simultaneously.

Mechanism. Third-degree heart block is the end result of progressively worsening second-degree AV block. Because a third-degree heart block can occur above or below the AV node, two different rhythms can take over. If it occurs above or at the crest of the AV node, a junctional rhythm will take over and drive the ventricles. The resulting QRS complexes will be narrow and occur at the intrinsic rate of the AV node (40 to 55 beats/minute). Whereas if the block occurs below the AV node, a ventricular pacemaker must take over. In such cases, the QRS complexes will be wide and at the intrinsic rate of the ventricular pacemaker (20 to 40 beats/minute).
Causes. Complete heart block is often the result of the same causes as Mobitz type 1 and Mobitz type 2. Other causes include inferior MI, degeneration of the conduction system, and AV-nodal blocking agents such as beta-blockers, non-dihydropyridine calcium channel blockers, adenosine, digitalis, and amiodarone.
Clinical significance. Patients with complete heart block are at great risk of developing asystole, ventricular tachycardia, and sudden cardiac death. Insertion of a permanent pacemaker is required.

Medical Management

In general, patients that present with first-degree or second-degree Mobitz type 1 AV block do not require treatment. Any provoking medications can be removed, and patients can be monitored on an outpatient basis. However, patients with higher degrees of AV block (Mobitz type 2 AV block, 3rd degree) tend to have severe damage to the conduction system. They are at a much greater risk of progressing into asystole, ventricular tachycardia, or sudden cardiac death. Hence, they require urgent admission for cardiac monitoring, backup temporary cardiac pacing, and insertion of a permanent pacemaker.[11][12][13][14]

Nursing Management

Monitor vital signs
Place the patient on a cardiac monitor
Obtain an ECG
Assess oxygenation
Check labs to ensure electrolytes are within normal limits
Place the patient at bed rest
Listen to the heart for murmurs
Monitor for fluid retention
Weigh patient
Hold medications that can disrupt cardiac rhythm (usually beta-blockers and antiarrhythmics)
If the patient is to have a pacemaker, educate the patient
Keep patient NPO
Call the pacemaker nurse after the pacemaker has been inserted
Educate patient about pacemakers, microwaves and TSA security checks which can cause magnetic interference with the pacemaker
After surgery, educate the patient to keep the arm still and avoid strenuous activity
Ask the patient to wear a medical alert bracelet
Tell the patient to inform the cardiologist before undergoing any type of procedure or surgery

When To Seek Help

Loss of consciousness
Altered mental status
Low blood pressure
Bradycardia

Outcome Identification

Prognosis depends on the various factors that include age and other chronic medical conditions such as diabetes mellitus, chronic kidney disease, underlying heart disease, and underlying types of AV block.

Coordination of Care

The management of heart block is best done with an interprofessional team because if the diagnosis is missed (esp higher degrees of heart block), the condition can have significant morbidity and mortality.

Except for a first-degree heart block, the rest of the patients should be referred to a cardiologist for a more definitive workup. Some of these patients may require a pacemaker which can be life-saving. Following treatment, the cardiology nurse should follow up on the patients to ensure that the heart rate has normalized and the patients have no symptoms.[15]

Anytime patients with a pacemaker undergo surgery, the cardiologist should be consulted first.

Health Teaching and Health Promotion

Patients with first-degree and asymptomatic Mobitz type 1 AV block usually can continue their usual activities but should be advised to avoid medications that can prolong the PR interval. Patients with Mobitz type 2 and third-degree AV block should discuss with their cardiologists about the need for pacemakers. All patients should be educated on alarming symptoms of hypoperfusion such as fatigue, lightheadedness, syncope, presyncope, or angina and seek timely medical treatment

(Click Image to Enlarge)

Figure.1. A 12-lead ECG shows constant, prolong PR interval (>200 milliseconds) with 1:1 association of P and QRS, consistent with first degree atrioventricular block.
Contributed by Intisar Ahmed

(Click Image to Enlarge)

Figure.2. An electrocardiogram shows progressive prolonging of PR interval followed by one blocked P wave, suggestive of second degree type I atrioventricular block (Wenckebach).
Contributed by Intisar Ahmed

(Click Image to Enlarge)

Figure.3. A 12-lead electrocardiogram shows constant PR interval of conducted P waves and a blocked P wave, happening at an unpredictable interval. These findings are consistent with Mobitz type II atrioventricular block.
Contributed by Intisar Ahmed

(Click Image to Enlarge)

Figure.4. A 12--lead ECG shows two consecutive blocked P waves followed by one conducted P wave, suggestive of high-grade atrioventricular block.
Contributed by Intisar Ahmed

(Click Image to Enlarge)

Figure.5. A 12-lead ECG shows P waves at a constant rate with no association of P and QRS complexes and the rate of QRS complex is slower than that of P waves. These findings are suggestive of third degree atrioventricular block.
Contributed by Intisar Ahmed

Details

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